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Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
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ZED Students POWER - Diclofenac is not a sweet 🍭. It is a potential poison when abused. Normal kidneys produce a hormone called prostaglandins that are used to protect the kidney from
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Vinpocetine reduces diclofenac-induced acute kidney injury through inhibition of oxidative stress, apoptosis, cytokine production, and NF-κB activation in mice - ScienceDirect
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Kidney damage from nonsteroidal anti‐inflammatory drugs—Myth or truth? Review of selected literature - Drożdżal - 2021 - Pharmacology Research & Perspectives - Wiley Online Library
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Biomedicines | Free Full-Text | A Single Oral Dose of Diclofenac Causes Transition of Experimental Subclinical Acute Kidney Injury to Chronic Kidney Disease
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Protective effect of curcumin on the kidney of diclofenac sodium-challenged mice: apoptotic, redox potential and histopathological outcomes | The Journal of Basic and Applied Zoology | Full Text
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A study on the toxic effect of different doses of Diclofenac sodium on the development of the kidney in the postnatal period | PDF
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